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Chronic glucolipotoxicity then promotes β-cell functional exhaustion and overt T2DM onset ( Throughout this process, hormones, inflammatory factors, and metabolites interact through a complex network, leading to a cascading failure of the metabolic network and jointly driving metabolic imbalance into a cyclical cascade reaction of “glucose and lipid accumulation → metabolic dysregulation → organ injury” ( However, this classical cascade view is insufficient to fully explain the high frequency of MAFLD and T2DM comorbidity