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The failure to note a clinical response to antioxidant therapy may be due to factors dependent upon dosage of, or interaction between, the antioxidant compounds given, or on restricted bioavailability of the antioxidants at critical anatomical sites, and does not invalidate the model of acute porphyria as a hyperoxidative condition per se ( Reference Thunell, Andersson and Harper 21 )

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Cystine is indispensable in TNBC growth and progression (27)