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In summary, GA inhibited the RhoA/CDC42 and PI3K-Akt-ERK1/2 pathways and activated the AMPK-eNOS-FAS signaling pathway, thereby suppressing the migration and proliferation of VSMCs (Figure 3)

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157 We recently provided in vitro evidence that both native and oxidized AAT control equally well ATP-induced IL-1β release from human mononuclear blood cells