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Blocking NNMT preserves the NAD+ precursor pool (1.2-1.6x increase in intracellular NAD+), increases SAM availability for epigenetic methylation, and activates SIRT1/SIRT3 signaling
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Animal and in vitro data suggest AOD-9604 does not meaningfully bind the GH receptor or stimulate IGF-1 production at the doses studied, which is considered a safety advantage over full-length GH
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