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stimulated TNF-α-induced human bronchial epithelial (BEAS-2B) cells with varying doses of cis-9, trans-11-conjugated linoleic acid (c9,t11-CLA) and demonstrated that c9,t11-CLA significantly inhibited the secretion of inflammatory cytokines (e.g., IL-6, IL-8, MCP-1, and CCL5) and reduced the expression level of intercellular adhesion molecule 1 (ICAM-1), thereby attenuating monocyte adhesion to inflamed bronchial epithelial cells (102)
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