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186 Its main biological effects are selective inhibition of PDE4, leading to increased cAMP levels in inflammatory cells, regulating the activity of inflammatory cells and regulating the release of inflammatory factors to exhibit anti-inflammatory effects

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It was found that the down-regulated expression of CC chemokine receptors 3 (CCR3), GATA binding proteins 1 (GATA-1), and GATA binding proteins 2 (GATA-2) could inhibit the differentiation of eosinophils both in vivo and in vitro , suggesting that exogenous IL-17 is most likely to prevent allergic airway inflammation by inhibiting eosinophil differentiation in the bone marrow ( It was also found that the elevated levels of Bcl-2 protein are responsible for the persistence of eosinophils and neutrophils during allergic airway inflammation Furthermore, the Bcl-2 inhibitor ABT-737 and ABT-199 may inhibit allergic airway inflammation by promoting the death of inflammatory cells, especially in asthma dominated by neutrophils and insensitive to corticosteroids (108)