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9,11 Pathomechanisms of Hashimoto thyroiditis The autoimmune presentation of HT is based on the interplay between environmental factors and genetic background, such as polymorphisms in human leukocyte antigen ( HLA ), T lymphocyte-associated 4 ( CTLA-4 ), protein tyrosine phosphatase, non-receptor type 22 ( PTPN22 ) genes, and X chromosome inactivation patterns, leading to an imbalance between self-tolerance mechanisms sustained by regulatory T and B lymphocytes

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These monotherapy results established proof of concept that amylin receptor agonism alone can produce clinically meaningful weight reduction and informed the dose selection of 2.4 mg for the combination product CagriSema