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For example, patients with heart failure must manage sodium and fluid intake to prevent decompensation, while individuals with neurodegenerative diseases often face dysphagia and altered nutrient absorption, requiring tailored dietary strategies [217]
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Nonetheless, when a cell (e.g., a hepatocyte) is exposed to inflammation or oxidative stress, NRF-2 has the capacity to escape from regulation by KEAP1 through the increase in the transcription of mRNA from NRF-2, with the objective of depleting the reserves of KEAP1, which cannot be recycled because of the suppression of another NRF-2 [21]
